Research Paper Volume 10, Issue 3 pp 371—385
CD40L promotes development of acute aortic dissection via induction of inflammation and impairment of endothelial cell function
Figure 3. Endothelial cells migration was significantly inhibited by CD40L. (A) Representative images showed different scratch area in groups incubated with or without recombinant human soluble CD40 ligand. (B) Quantitative analysis showed significantly larger scratch area in wound area incubated with CD40L. Data were expressed as percentage ± SD. **P < 0.05.