Research Paper Volume 13, Issue 11 pp 15013—15031

Figure 5. CG9911 modulates intracellular calcium homeostasis. (A) Fluorescence changes of w¹¹¹⁸ and CG9911 mutant fat bodies which were treated with 10μM inomycin. Scale bar = 50 μm. (B) The dynamic changes of fluorescence intensity(ΔF/F₀) in the signal fat cell of w¹¹¹⁸ and CG9911 mutant files when stimulated with 10μM inomycin. (C) The difference of mean F/F₀ between w¹¹¹⁸ and CG9911 mutant fat cells when treated with 10μM inomycin. Data are presented as the means ± s.e.m; ** p<0.01. (D) The comparation of the mean data for time from basal to peak (t_max), half time raise (t_1/2 on), and decay (t_1/2 off) between w¹¹¹⁸ and CG9911 mutant fat cells when treated with 10μM inomycin. Data are presented as the means ± s.e.m; ** p<0.01, *** p<0.001. (E) Ca²⁺ imaging of w¹¹¹⁸ and CG9911 mutant fat bodies when treated with 10 μM TG. (F) The dynamic changes of fluorescence intensity(ΔF/F₀) in the signal fat cell of w¹¹¹⁸ and CG9911 mutant files when stimulated with 10μM TG. (G) The difference of mean data of F/F₀ between w¹¹¹⁸ and CG9911 mutant fat bodies when treated with 10 μM TG. Data are presented as the means ± s.e.m; *** p<0.001. (H) The comparation of the mean data for time from basal to peak (t_max), half time raise (t_1/2 on), and decay (t_1/2 off) between w¹¹¹⁸ and CG9911 mutant fat cells when treated with 10μM TG. Data are presented as the means ± s.e.m; *p<0.05. (I) The quiescent Ca²⁺ level in the signal fat cell of w¹¹¹⁸ and CG9911 mutant files.